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Adam Beach

Concordia University

33

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Mechanisms through which lithocholic acid delays yeast chronological aging under caloric restriction conditions

2018 Oncotarget
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Caloric restriction delays yeast chronological aging by remodeling carbohydrate and lipid metabolism, altering peroxisomal and mitochondrial functionalities, and postponing the onsets of apoptotic and liponecrotic modes of regulated cell death

2018 Oncotarget
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Lithocholic bile acid accumulated in yeast mitochondria orchestrates a development of an anti-aging cellular pattern by causing age-related changes in cellular proteome

2015 Cell Cycle
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Mechanisms by Which Different Functional States of Mitochondria Define Yeast Longevity

2015 International Journal of Molecular Sciences
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Macromitophagy, neutral lipids synthesis, and peroxisomal fatty acid oxidation protect yeast from "liponecrosis", a previously unknown form of programmed cell death.

2014 Cell Cycle
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Ring-substituted analogs of 3,3′-diindolylmethane (DIM) induce apoptosis and necrosis in androgen-dependent and –independent prostate cancer cells

2014 Investigational New Drugs
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Mitochondrial membrane lipidome defines yeast longevity (956.2)

2014 The FASEB Journal
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Mechanism of liponecrosis, a distinct mode of programmed cell death

2014 Cell Cycle
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Macromitophagy is a longevity assurance process that in chronologically aging yeast limited in calorie supply sustains functional mitochondria and maintains cellular lipid homeostasis (953.3)

2014 The FASEB Journal
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Bile acids induce apoptosis selectively in androgen-dependent and -independent prostate cancer cells

2013 PeerJ
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Citations (53)