Evaluation of disease burden of hepatitis C in China

s / Journal of Clinical Virology 69 (2015) 223–246 231 the antiviral activity of type III IFNs in vivo. The exposure of newly discovered IFN4 to macrophages and dendritic cells also raised the expression of its own receptor, which shows that expression of IFN4 protein in patients with hepatitis C might augment type I IFN treatment and help to lower viral titres. Interpretation: The results of this study may help us to understand the mechanisms involved in the selective expression of IFNLR1 and exceptions involved. http://dx.doi.org/10.1016/j.jcv.2015.06.029