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Edwige Belotti
Edwige Belotti
Claude Bernard University Lyon 1
Cell biology
Biology
Mechanistic target of rapamycin
PI3K/AKT/mTOR pathway
Internal medicine
4
Papers
44
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H2A.Z is dispensable for both basal and activated transcription in post-mitotic mouse muscles.
2020
Nucleic Acids Research
Edwige Belotti
Nicolas Lacoste
Thomas Simonet
Christophe Papin
Kiran Padmanabhan
Isabella Scionti
Yann-Gaël Gangloff
Lorrie Ramos
Defne Dalkara
Ali Hamiche
Stefan Dimitrov
Laurent Schaeffer
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Citations (13)
H2A.Z is dispensable for both basal and activated transcription in post-mitotic mouse muscles
2019
bioRxiv
Edwige Belotti
Nicolas Lacoste
Thomas Simonet
Christophe Papin
Kiran Padmanabhan
Lorrie Ramos
Defne Dalkara
Isabella Scionti
Ali Hamiche
Stefan Dimitrov
Laurent Schaeffer
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mTOR inactivation in myocardium from infant mice rapidly leads to dilated cardiomyopathy due to translation defects and p53/JNK-mediated apoptosis.
2016
Journal of Molecular and Cellular Cardiology
Laetitia Mazelin
Baptiste Panthu
Anne-Sophie Nicot
Edwige Belotti
Lionel Tintignac
Geoffrey Teixeira
Qing Zhang
Valérie Risson
Dominique Baas
Emilie Delaune
Geneviève Derumeaux
Daniel Taillandier
Théophile Ohlmann
Michel Ovize
Yann-Gaël Gangloff
Laurent Schaeffer
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Citations (31)
0452 : MTOR inactivation during early postnatal development of mice myocardium leads to severe dilated cardiomyopathy due to altered translational efficiency and hypoxia-induced apoptosis
2015
Archives of Cardiovascular Diseases Supplements
Laetitia Mazelin Bowyer
Anne-Sophie Nicot
Emilie Delaune
Ging Zhang
Edwige Belotti
Valérie Risson
Dominique Baas
Yann Gael Gangloff
Laurent Schaeffer
Baptiste Panthu
Théophile Ohlmann
Geoffrey Texeira
Michel Ovize
Geneviève Derumeaux
Daniel Taillandier
Lionel Tintignac
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