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Zehra Kevser Timur
Zehra Kevser Timur
İzmir Institute of Technology
Biology
Biochemistry
Ganglioside
Sialic acid
Tay-Sachs disease
6
Papers
53
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Sialidase neu4 deficiency is associated with neuroinflammation in mice.
2021
Glycoconjugate Journal
Zehra Kevser Timur
Orhan Kerim Inci
Secil Akyildiz Demir
Volkan Seyrantepe
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GM2 ganglioside accumulation causes neuroinflammation and behavioral alterations in a mouse model of early onset Tay-Sachs disease
2020
Journal of Neuroinflammation
Secil Akyildiz Demir
Zehra Kevser Timur
Nurselin Ates
Luis Alarcón-Martínez
Volkan Seyrantepe
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Citations (3)
Murine Sialidase Neu3 facilitates GM2 degradation and bypass in mouse model of Tay-Sachs disease
2018
Experimental Neurology
Volkan Seyrantepe
Secil Akyildiz Demir
Zehra Kevser Timur
Johanna von Gerichten
Christian Marsching
Esra Erdemli
Emin Oztas
Kohta Takahashi
Kazunori Yamaguchi
Nurselin Ates
Buket Dönmez-Demir
Turgay Dalkara
Katrin Erich
Carsten Hopf
Roger Sandhoff
Taeko Miyagi
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Citations (27)
Lysosomal Cathepsin A Plays a Significant Role in the Processing of Endogenous Bioactive Peptides.
2016
Frontiers in Molecular Biosciences
Zehra Kevser Timur
Secil Akyildiz Demir
Volkan Seyrantepe
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Citations (15)
Neuraminidase-1 contributes significantly to the degradation of neuronal B-series gangliosides but not to the bypass of the catabolic block in Tay-Sachs mouse models.
2015
Molecular genetics and metabolism reports
Zehra Kevser Timur
S. Akyildiz Demir
Christian Marsching
Roger Sandhoff
Volkan Seyrantepe
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Citations (8)
Mass spectrometry and thin layer chromatography analysis of brain gangliosides in Tay-Sachs disease mouse models
2014
Molecular Genetics and Metabolism
Volkan Seyrantepe
Zehra Kevser Timur
Christian Marsching
Roger Sandhoff
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