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Oliver Schulz

Francis Crick Institute
London Research Institute

32

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Publisher Correction: The receptor DNGR-1 signals for phagosomal rupture to promote cross-presentation of dead-cell-associated antigens.

2021 Nature Immunology
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The receptor DNGR-1 signals for phagosomal rupture to promote cross-presentation of dead-cell-associated antigens.

2021 Nature Immunology
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Secreted gelsolin inhibits DNGR-1-dependent cross-presentation and cancer immunity.

2021 Cell
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AGI-134: a fully synthetic α-Gal glycolipid that converts tumors into in situ autologous vaccines, induces anti-tumor immunity and is synergistic with an anti-PD-1 antibody in mouse melanoma models

2019 Cancer Cell International
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Direct reprogramming of fibroblasts into antigen-presenting dendritic cells

2018 Science immunology
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Myosin II Synergizes with F-Actin to Promote DNGR-1-Dependent Cross-Presentation of Dead Cell-Associated Antigens

2018 Cell Reports
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DNGR-1 in dendritic cells limits tissue damage by dampening neutrophil recruitment

2018 Science
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Abstract 616: The novel α-Gal-based immunotherapy AGI-134 invokes CD8+ T cell-mediated immunity by driving tumor cell destruction, phagocytosis and tumor-associated antigen cross-presentation via multiple antibody-mediated effector functions

2017 Cancer Research
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A pH‐ and ionic strength‐dependent conformational change in the neck region regulates DNGR‐1 function in dendritic cells

2016 The EMBO Journal
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RIPK1 and NF-κB signaling in dying cells determines cross-priming of CD8 + T cells

2015 Science
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Citations (272)