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Kirit Pindolia

Henry Ford Health System
Wayne State University

27

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477

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Corrigendum to “First contiguous gene deletion causing biotinidase deficiency: The enzyme deficiency in three Sri Lankan children” [Mol. Genet. Metab. Rep. 2 (2016) 81–84]

2017 Molecular genetics and metabolism reports
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Developmental window of sensorineural deafness in biotinidase-deficient mice

2017 Journal of Inherited Metabolic Disease
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Differential gene expression during early development in brains of wildtype and biotinidase-deficient mice

2016 Molecular genetics and metabolism reports
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Inhibition of hTERT/telomerase contributes to the antitumor activity of pristimerin in pancreatic ductal adenocarcinoma cells

2015 Oncology Reports
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First contiguous gene deletion causing biotinidase deficiency: The enzyme deficiency in three Sri Lankan children

2015 Molecular genetics and metabolism reports
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Role of telomerase in anticancer activity of pristimerin in prostate cancer cells.

2015 Journal of Experimental Therapeutics and Oncology
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Pristimerin, a quinonemethide triterpenoid, induces apoptosis in pancreatic cancer cells through the inhibition of pro-survival Akt/NF-κB/mTOR signaling proteins and anti-apoptotic Bcl-2

2014 International Journal of Oncology
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Ubiquitin-proteasomal degradation of antiapoptotic survivin facilitates induction of apoptosis in prostate cancer cells by pristimerin

2014 International Journal of Oncology
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Induction of Apoptosis in Pancreatic Cancer Cells by CDDO-Me Involves Repression of Telomerase through Epigenetic Pathways.

2014 Journal of carcinogenesis & mutagenesis
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Characterization and functional analysis of cellular immunity in mice with biotinidase deficiency.

2014 Molecular Genetics and Metabolism
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