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Mukarram El-Banna

Center for Advanced Biotechnology and Medicine

9

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Analysis of brain and cerebrospinal fluid from mouse models of the three major forms of neuronal ceroid lipofuscinosis reveals changes in the lysosomal proteome

2019 Molecular & Cellular Proteomics
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Lysosomal enzyme tripeptidyl peptidase 1 plays a role in degradation of beta amyloid fibrils

2019
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Inducible transgenic expression of tripeptidyl peptidase 1 in a mouse model of late-infantile neuronal ceroid lipofuscinosis

2018 PLOS ONE
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Extending the mannose-6 phosphate glycoproteome by high resolution/accuracy mass spectrometry analysis of control and acid phosphatase 5-deficient mice.

2013 Molecular & Cellular Proteomics
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Large-volume Intrathecal Enzyme Delivery Increases Survival of a Mouse Model of Late Infantile Neuronal Ceroid Lipofuscinosis

2011 Molecular Therapy
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Residual levels of tripeptidyl-peptidase I activity dramatically ameliorate disease in late-infantile neuronal ceroid lipofuscinosis.

2008 Molecular Genetics and Metabolism
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427. AAV2- and AAV5-Mediated CNS Delivery of Human CLN2 Reduces Lysosomal Storage in a Mouse Model of Late Infantile Neuronal Ceroid Lipofuscinosis

2005 Molecular Therapy
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Genetic evidence for nonredundant functional cooperativity between NPC1 and NPC2 in lipid transport

2004 Proceedings of the National Academy of Sciences of the United States of America
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A Mouse Model of Classical Late-Infantile Neuronal Ceroid Lipofuscinosis Based on Targeted Disruption of the CLN2 Gene Results in a Loss of Tripeptidyl-Peptidase I Activity and Progressive Neurodegeneration

2004 The Journal of Neuroscience
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