Metabolic Syndrome Mediates ROS-miR-193b-NFYA-Dependent Downregulation of Soluble Guanylate Cyclase and Contributes to Exercise-Induced Pulmonary Hypertension in Heart Failure With Preserved Ejection Fraction.

Taijyu Satoh,Longfei Wang,Cristina Espinosa-Diez,Bing Wang,Scott A. Hahn,Kentaro Noda,Elizabeth R. Rochon,Matthew R. Dent,Andrea R. Levine,Jeffrey J. Baust,Samuel K. Wyman,Yijen L. Wu,Georgios Triantafyllou,Ying Tang,Mike Reynolds,Sruti Shiva,Cynthia St. Hilaire,Delphine Gomez,Dmitry A. Goncharov,Elena A. Goncharova,Stephen Y. Chan,Adam C. Straub,Yen-Chun Lai,Charles F. McTiernan,Mark T. Gladwin

Metabolic Syndrome Mediates ROS-miR-193b-NFYA-Dependent Downregulation of Soluble Guanylate Cyclase and Contributes to Exercise-Induced Pulmonary Hypertension in Heart Failure With Preserved Ejection Fraction.

2021

Background: Many patients with heart failure with preserved ejection fraction have metabolic syndrome and develop exercise-induced pulmonary hypertension (EIPH). Increases in pulmonary vascular res...

Keywords:

Downregulation and upregulation
Internal medicine
Heart failure with preserved ejection fraction
Guanylate cyclase
Medicine
Metabolic syndrome
Endocrinology
Nitric oxide
Pulmonary hypertension
mir 193b

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