A common Chk1-dependent phenotype of DNA double-strand break suppression in two distinct radioresistant cancer types
2019
Purpose
Triple-negative breast cancers (TNBC) are often resistant to treatment with ionizing radiation (IR). We sought to investigate whether pharmacologic inhibition of Chk1 kinase, which is commonly overexpressed in TNBC, preferentially sensitizes TNBC cells to IR.
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