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David W. Rowe

University of Connecticut Health Center
University of Connecticut

304

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14125

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GNAS Heterozygous Inactivation Differentially Affects Osteoclast-Specific Calcitonin Receptor Bioactivity in a Mouse Model of Albright Hereditary Osteodystrophy Based Upon Parental Inheritance

2021 Journal of the Endocrine Society
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Senolytics improve bone forming potential of bone marrow mesenchymal stem cells from aged mice.

2021 npj Regenerative Medicine
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Parental origin of Gsα inactivation differentially affects bone remodeling in a mouse model of Albright hereditary osteodystrophy

2021 bioRxiv
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Author response for "Parental origin of Gsα inactivation differentially affects bone remodeling in a mouse model of Albright hereditary osteodystrophy"

2021
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Parental origin of Gsα inactivation differentially affects bone remodeling in a mouse model of Albright hereditary osteodystrophy

2021
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Biodegradable nanofiber bone-tissue scaffold as remotely-controlled and self-powering electrical stimulator

2020 Nano Energy
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Transplanting cells from old but not young donors causes physical dysfunction in older recipients

2020 Aging Cell
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P7. Hierarchical surface roughness produced with additive manufacturing technology significantly increases osteogenic cellular differentiation and gene expression when compared to PEEK and smooth titanium surfaces

2020 The Spine Journal
- David W. Rowe
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cTAP: A Machine Learning Framework for Predicting Target Genes of a Transcription Factor using a Cohort of Gene Expression Data Sets

2020 BIBM | Bioinformatics and Biomedicine
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Skeletal Screening IMPC/KOMP using μCT and Computer Automated Cryohistology: Application to the Efna4 KO mouse line.

2020 Bone
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