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David M. Findlay

University of Adelaide
St. Vincent's Institute of Medical Research
Royal Adelaide Hospital

339

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Elevated levels of active Transforming Growth Factor β1 in the subchondral bone relate spatially to cartilage loss in human knee osteoarthritis

2021 Bone reports
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Greater heterogeneity of the bone mineralisation density distribution and low bone matrix mineralisation characterise tibial subchondral bone marrow lesions in knee osteoarthritis patients.

2021 Bone
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A case of autosomal dominant osteopetrosis type II with a severe bone phenotype but no amino acid converting mutation in the CLCN7 gene

2021 medRxiv
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Elevated levels of active transforming growth factor β1 in the subchondral bone relate spatially to impaired bone quality and cartilage loss in human knee osteoarthritis

2021 Osteoarthritis and Cartilage
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Elevated levels of active Transforming Growth Factor β1 in the subchondral bone relate spatially to cartilage loss and impaired bone quality in human knee osteoarthritis

2021 bioRxiv
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Raman microspectroscopy demonstrates reduced mineralization of subchondral bone marrow lesions in knee osteoarthritis patients

2020 Bone reports
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THE OSTEOCYTE AS A TARGET CELL AND DISEASE NIDUS IN HUMAN PERIPROSTHETIC JOINT INFECTION

2020
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Bone Marrow Lesions in Knee Osteoarthritis: Regional Differences in Tibial Subchondral Bone Microstructure and their Association with Cartilage Degeneration

2019 Osteoarthritis and Cartilage
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The osteocyte: master directors of bone remodelling, mineralisation and phosphate-calcium homeostasis

2019
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Osteocytes respond to particles of clinically-relevant conventional and cross-linked polyethylene and metal alloys by up-regulation of resorptive and inflammatory pathways

2019 Acta Biomaterialia
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