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Joshua K. Stone

University of South Alabama

12

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145

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SON drives oncogenic RNA splicing in glioblastoma by regulating PTBP1/PTBP2 switching and RBFOX2 activity.

2021 Nature Communications
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eNEMAL, an enhancer RNA transcribed from a distal MALAT1 enhancer, promotes NEAT1 long isoform expression.

2021 PLOS ONE
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SON inhibits megakaryocytic differentiation via repressing RUNX1 and the megakaryocytic gene expression program in acute megakaryoblastic leukemia.

2020 Cancer Gene Therapy
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SON haploinsufficiency causes impaired pre-mRNA splicing of CAKUT genes and heterogeneous renal phenotypes

2019 Kidney International
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Hypoxia induces cancer cell-specific chromatin interactions and increases MALAT1 expression in breast cancer cells

2019 Journal of Biological Chemistry
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Abstract LB-204: SON controls the oncogenic alternative splicing program in glioblastoma by regulating PTBP1/2 switch and RBFOX2 activity

2018 Cancer Research
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Abstract 1479: Hypoxia upregulates the long noncoding RNA MALAT1 through increased chromatin looping

2018 Cancer Research
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Abstract 1978: SON and its splice variants regulate MLL1/2 complex-mediated H3K4me3 and transcription of leukemia-associated genes

2016 Cancer Research
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SON and Its Alternatively Spliced Isoforms Control MLL Complex-Mediated H3K4me3 and Transcription of Leukemia-Associated Genes

2016 Molecular Cell
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Abstract 1835: NFATC1 regulates the long non-coding RNA MALAT1 in breast cancer

2016 Cancer Research
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