CLINICAL-LIVER, PANCREAS, AND BILIARY TRACT

(HR, 0.37; 95% CI, 0.22‐0.62). Diagnostic capacity of HVPG was greater than for MELD or Child‐Pugh score. Conclusions: HVPG, MELD, and albumin independently predict clinical decompensation in patients with compensated cirrhosis. Patients with an HVPG <10 mm Hg have a 90% probability of not developing clinical decompensation in a median follow-up of 4 years.