[Acute polyradiculoneuropathy of Guillain-Barré. Critical review of potential etiopathogeneses].

: The authors go over the etiopathogenetic hypothesis suggested, in the last years, a propos of the Guillain-Barre syndrome. They point out that about two/thirds of cases are preceded by infectious symptoms of the upper respiratory and alimentary tracts, with prevailing viral etiology. The nervous lesion, which characterize the histology of the illness, seems related to immunological change. In particular one must emphasize the possibility that the Guillain-Barre syndrome is an autoimmune disorder of delayed hypersensivity, the laboratoristics model of which is constituted from experimental allergic neuritis.