Pulmonary vasodilation in the rat by insulin in vitro could indicate potential hazard for inhaled insulin
2003
Aims/hypothesis
Hypoxic pulmonary vasoconstriction is an essential mechanism to prevent hypoxaemia in lung diseases. Insulin is known to be a systemic vasodilator but its effect on the pulmonary circulation is not known. Inhaled particulate insulin can generate locally high concentrations in the lung which could be physiologically important.
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