PHARMACOGENOMIC IMPLICATIONS OF RENIN ANGIOTENSIN ALDOSTERONE SYSTEM INHIBITION IN ST-ELEVATION MYOCARDIAL INFARCTION

Background: A complex network of gene-environment interactions may be related to myocardial infarction. To date, several single nucleotide polymorphisms (SNPs) have been investigated as potential determinants in terms of predisposition, prognosis and response to therapy, but with no definite